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dc.contributor.author Mur, Luis A. J.
dc.contributor.author Kenton, Paul
dc.contributor.author Lloyd, Amanda J.
dc.contributor.author Ougham, Helen J.
dc.contributor.author Prats, Elena
dc.date.accessioned 2009-05-21T12:53:36Z
dc.date.available 2009-05-21T12:53:36Z
dc.date.issued 2007-12-12
dc.identifier.citation Mur , L A J , Kenton , P , Lloyd , A J , Ougham , H J & Prats , E 2007 , ' The hypersensitive response; the centenary is upon us but how much do we know? ' Journal of Experimental Botany , vol 59 , no. 3 , pp. 501-520 . , 10.1093/jxb/erm239 en
dc.identifier.issn 1460-2431
dc.identifier.other PURE: 103119
dc.identifier.other dspace: 2160/2336
dc.identifier.uri http://hdl.handle.net/2160/2336
dc.description Mur, L. A., Kenton, P., Lloyd, A. J., Ougham, H. J., Prats, E. (2008). The hypersensitive response: the centenary is upon us but how much do we know?  Journal of Experimental Botany, 59, (3), 501-520. Modelling Plant Systems. SEB Annual meeting, Glasgow, UK, 28-29 June 2007. Sponsorship: BBSRC: Royal Society : Aberystwyth University Research Fund: EU Human Resources Mobility Fund: ERG : Spanish Science and Technology Ministry. On file IMPF: 04.00 RONO: 1310 3001 en
dc.description.abstract With the centenary of the first descriptions of ‘hypersensitiveness’ following pathogenic challenge upon us, it is appropriate to assess our current understanding of the hypersensitive response (HR) form of cell death. In recent decades our understanding of the initiation, associated signalling, and some important proteolytic events linked to the HR has dramatically increased. Genetic approaches are increasingly elucidating the function of the HR initiating resistance genes and there have been extensive analyses of death-associated signals, calcium, reactive oxygen species (ROS), nitric oxide, salicylic acid, and now sphingolipids. At the same time, attempts to draw parallels between mammalian apoptosis and the HR have been largely unsuccessful and it may be better to consider the HR to be a distinctive form of plant cell death. We will consider if the HR form of cell death may occur through metabolic dysfunction in which malfunctioning organelles may play a major role. This review will highlight that although our knowledge of parts of the HR is excellent, a comprehensive molecular model is still to be attained. en
dc.format.extent 20 en
dc.language.iso eng
dc.relation.ispartof Journal of Experimental Botany en
dc.subject pathogen en
dc.subject hypersensitive response en
dc.subject programmed cell death en
dc.subject resistance en
dc.title The hypersensitive response; the centenary is upon us but how much do we know? en
dc.type Text en
dc.type.publicationtype Article (Journal) en
dc.identifier.doi http://dx.doi.org/10.1093/jxb/erm239
dc.contributor.institution Institute of Biological, Environmental and Rural Sciences en
dc.description.status Peer reviewed en


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